1·Pain and hyperalgesia was caused by injury of tissue and peripheral nerves.
组织损伤及外周神经损伤后可导致痛及痛过敏。
2·RESULTS: Thermal hyperalgesia developed between Days 12 and 18 after cancer cell inoculation.
结果：热痛觉过敏在肿瘤细胞植入后的12 - 18天发生进展。
3·Conclusion There is a close relationship between epidural scar adhesion and thermal hyperalgesia.
结论硬膜外瘢痕粘连重，则痛阈降低。热敏实验与硬膜外瘢痕粘连有密切关系。
4·All these results suggested that VR1 is critical for the expression of inflammation-induced heat hyperalgesia.
这些结果进一步说明VR1在炎症性热痛敏的形成中起着非常重要的作用。
5·Many researchers, therefore, have amelioration of hyperalgesia and allodynia foremost in their minds as they hunt for new analgesics.
因此，许多研究人员在寻求新的止痛药方时，都会把解除痛觉过敏及异位疼痛列入优先考量。
6·The behavior results showed that blocking peripheral 5-HT_(2A)receptors inhibited the thermal hyperalgesia in inflammatory and neuropathic pain.
行为学结果显示，阻断外周5-HT_（2A）受体能够明显抑制慢性炎症及神经病理性痛引发的热痛觉过敏。
7·Results Sprout number of DRG in experimental group rats decreased obviously and the symptom of hyperalgesia was improved as compared to control rats.
结果实验组大鼠drg内交感芽生明显减少，热痛过敏症状被抑制。
8·The peripheral nerve injury caused by some pathological factors results in pathological chronic pain, such as hyperalgesia, allodynia, and spontaneous pain ect.
各种因素引起的外周神经损伤可诱发痛觉过敏、感觉倒错等病理现象。
9·Preemptive analgesia is one of the analgesia measures which decreases the incidence of hyperalgesia and allodynia by reducing peripheral and central sensitization.
超前镇痛是通过防止外周和中枢敏化来降低伤害性刺激引起的痛觉过敏和痛觉异常的一种镇痛方法。
10·NGF levels are increased in inflammatory processes and administration of exogenous NGF leads to hyperalgesia, hypersensitivity to thermal stimulation and muscular pain.
神经生长因子水平炎症过程中的神经生长因子增加外源性和管理导致痛觉过敏，过敏热刺激和肌肉痛。