内皮素（ET）是一种血管收缩肽，与ET受体结合而发挥作用。
目前用于治疗原发性高血压的药物中有几种是通过抑制交感性血管收缩的紧张度而起作用的。
医生已经指示一个血管收缩剂，它会加在点滴里让出血停止。
高脂高糖饮食诱导灵长目动物颅内动脉粥样硬化并增强血管收缩反应
1·The results show; (1) The increased hepatic vasoconstrictor reactivity and the adhesive function of platelets in viral infection are the basic mechanism of the hepatic microcirculatory disturbance.
结果提示：（1）在病毒感染中肝血管收缩反应性上升及血小板粘附功能增强是肝微循环障碍发生的基础。
2·Conclusion CRA seems to be relate to the renal and splenic hemodynamics, also relate to the histology, the Doppler parameters of portal hypertension and the activity of vasoconstrictor systems.
结论视网膜中动脉可能与肾脏、脾脏的血流动力学变化有关，同样与组织学、门脉高压多普勒参数和血管收缩系统的活性有关。
3·Endothelin (ET) is a recently discovered potent vasoconstrictor peptide exerting effects through combination with its receptor.
内皮素(et)是一种血管收缩肽，与et受体结合而发挥作用。
1·Endothelin-1(ET-1) is a powerful intrinic vasoconstrictor, which plays an important role in the development of pulmonary hypertension.
内皮素（ET）-1是强有力的内源性血管收缩剂，在肺动脉高压的发病机制中发挥重要作用。